PM2.5-induced adipose tissue dysfunction can trigger metabolic disturbances

Andrew Shin, Lucio Della Guardia

Research output: Contribution to journalArticlepeer-review

4 Scopus citations

Abstract

Exposure to particulate matter ≤2.5 μm in diameter (PM2.5) alters cardiometabolic homeostasis. The reduced oxidative capacity in brown adipocytes and the development of inflammation and insulin resistance in white adipose tissue (WAT) can account for the dysmetabolic setting on PM2.5 exposure. In this forum article, we discuss relevant evidence to highlight a causal connection between PM2.5-induced adipose tissue dysfunction and cardiometabolic disturbances.

Original languageEnglish
Pages (from-to)737-740
Number of pages4
JournalTrends in Endocrinology and Metabolism
Volume33
Issue number11
DOIs
StatePublished - Nov 2022

Keywords

  • adipocytes
  • air pollution
  • cardiovascular
  • inflammation
  • insulin resistance
  • particulate matter

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