Nicotine-induced oxidative stress contributes to EMT and stemness during neoplastic transformation through epigenetic modifications in human kidney epithelial cells.

Yu Wei Chang; Kamaleshwar Singh

Nicotine-induced oxidative stress contributes to EMT and stemness during neoplastic transformation through epigenetic modifications in human kidney epithelial cells.

Environmental Toxicology

Research output: Contribution to journal › Article › peer-review

Original language	English
Pages (from-to)	65-76
Journal	Toxicology and Applied Pharmacology
State	Published - Apr 29 2019

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@article{738c70ef28dc446eb6952bf609b9546e,

title = "Nicotine-induced oxidative stress contributes to EMT and stemness during neoplastic transformation through epigenetic modifications in human kidney epithelial cells.",

author = "Chang, {Yu Wei} and Kamaleshwar Singh",

year = "2019",

month = apr,

day = "29",

language = "English",

pages = "65--76",

journal = "Toxicology and Applied Pharmacology",

}

TY - JOUR

T1 - Nicotine-induced oxidative stress contributes to EMT and stemness during neoplastic transformation through epigenetic modifications in human kidney epithelial cells.

AU - Chang, Yu Wei

AU - Singh, Kamaleshwar

PY - 2019/4/29

Y1 - 2019/4/29

M3 - Article

SP - 65

EP - 76

JO - Toxicology and Applied Pharmacology

JF - Toxicology and Applied Pharmacology

ER -

Nicotine-induced oxidative stress contributes to EMT and stemness during neoplastic transformation through epigenetic modifications in human kidney epithelial cells.

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