Chronic exposure to arsenic causes increased cell survival, DNA damage and increased expression of mitochondrial transcription factor A (mtTFA) in human prostate epithelial cells.

Kamaleshwar Singh; Ragini Kumari; Justin Treas; James W DuMond

Chronic exposure to arsenic causes increased cell survival, DNA damage and increased expression of mitochondrial transcription factor A (mtTFA) in human prostate epithelial cells.

Kamaleshwar Singh, Ragini Kumari, Justin Treas, James W DuMond

Environmental Toxicology

Research output: Contribution to journal › Article › peer-review

Original language	English
Pages (from-to)	340-349
Journal	Chemical Research in Toxicology
State	Published - Jan 14 2011

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@article{dcd553805b22412aba464d8df4d05db4,

title = "Chronic exposure to arsenic causes increased cell survival, DNA damage and increased expression of mitochondrial transcription factor A (mtTFA) in human prostate epithelial cells.",

author = "Kamaleshwar Singh and Ragini Kumari and Justin Treas and DuMond, {James W}",

year = "2011",

month = jan,

day = "14",

language = "English",

pages = "340--349",

journal = "Chemical Research in Toxicology",

publisher = "ACS",

}

Chronic exposure to arsenic causes increased cell survival, DNA damage and increased expression of mitochondrial transcription factor A (mtTFA) in human prostate epithelial cells. / Singh, Kamaleshwar; Kumari, Ragini; Treas, Justin; DuMond, James W.

In: Chemical Research in Toxicology, 14.01.2011, p. 340-349.

Research output: Contribution to journal › Article › peer-review

TY - JOUR

T1 - Chronic exposure to arsenic causes increased cell survival, DNA damage and increased expression of mitochondrial transcription factor A (mtTFA) in human prostate epithelial cells.

AU - Singh, Kamaleshwar

AU - Kumari, Ragini

AU - Treas, Justin

AU - DuMond, James W

PY - 2011/1/14

Y1 - 2011/1/14

M3 - Article

SP - 340

EP - 349

JO - Chemical Research in Toxicology

JF - Chemical Research in Toxicology

ER -

Chronic exposure to arsenic causes increased cell survival, DNA damage and increased expression of mitochondrial transcription factor A (mtTFA) in human prostate epithelial cells.

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